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faktor IX

Storage-stable blood coagulation factor acting in the intrinsic pathway. Its activated form, IXa, forms a complex with factor VIII and calcium on platelet factor 3 to activate factor X to Xa. Deficiency of factor IX results in HEMOPHILIA B (Christmas Disease).
MSH

Factor IX is a vitamin k-dependent plasma protein that participates in the intrinsic pathway of blood coagulation by converting factor X to its active form in the presence of ca(2+) ions, phospholipids, and factor VIIIa.
NCI

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faktor IXa

Activated form of factor IX. This activation can take place via the intrinsic pathway by the action of factor XIa and calcium, or via the extrinsic pathway by the action of factor VIIa, thromboplastin, and calcium. Factor IXa serves to activate factor X to Xa by cleaving the arginyl-leucine peptide bond in factor X.
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faktor V

Heat- and storage-labile plasma glycoprotein which accelerates the conversion of prothrombin to thrombin in blood coagulation. Factor V accomplishes this by forming a complex with factor Xa, phospholipid, and calcium (prothrombinase complex). Deficiency of factor V leads to Owren`s disease.
MSH

heat and storage labile plasma glycoprotein which accelerates the conversion of prothrombin to thrombin in blood coagulation by forming a complex with factor Xa, phospholipid, and calcium (prothrombinase complex); deficiency of factor V leads to Owrens disease.
CSP

a factor in the clotting of blood
CHV

Coagulation factor V (2224 aa, ~252 kDa) is encoded by the human F5 gene. This protein plays a role in the coagulation cascade.
NCI

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faktor V – nedostatek

A deficiency of blood coagulation factor V (known as proaccelerin or accelerator globulin or labile factor) leading to a rare hemorrhagic tendency known as Owren`s disease or parahemophilia. It varies greatly in severity. Factor V deficiency is an autosomal recessive trait. (Dorland, 27th ed)
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faktor Va

Activated form of factor V. It is an essential cofactor for the activation of prothrombin catalyzed by factor Xa.
MSH

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faktor VII

Heat- and storage-stable plasma protein that is activated by tissue thromboplastin to form factor VIIa in the extrinsic pathway of blood coagulation. The activated form then catalyzes the activation of factor X to factor Xa.
MSH

A clotting factor found in the serum. It combines with tissue factor (factor III) to activate factor X in the clotting pathway.
NCI

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faktor VII – nedostatek

An autosomal recessive characteristic or a coagulation disorder acquired in association with VITAMIN K DEFICIENCY. FACTOR VII is a Vitamin K dependent glycoprotein essential to the extrinsic pathway of coagulation.
MSH

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faktor VIIa

Activated form of factor VII. Factor VIIa activates factor X in the extrinsic pathway of blood coagulation.
MSH

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faktor VIII

Blood-coagulation factor VIII. Antihemophilic factor that is part of the factor VIII/von Willebrand factor complex. Factor VIII is produced in the liver and acts in the intrinsic pathway of blood coagulation. It serves as a cofactor in factor X activation and this action is markedly enhanced by small amounts of thrombin.
MSH

antihemophilic factor that is part of the factor VIII/von Willebrand factor complex; produced in the liver and acts in the intrinsic pathway of blood coagulation; serves as a cofactor in factor X activation and this action is markedly enhanced by small amounts of thrombin.
CSP

Human coagulation factor-VIII (2351 aa, 267 kDa precursor) is encoded by human factor eight (F8) gene. The protein is an extracellular anti-hemophilic factor that is part of the von Willebrand factor complex. The protein plays a role in the blood coagulation pathway.
NCI

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faktor VIIIa

Activated form of factor VIII. The B-domain of factor VIII is proteolytically cleaved by thrombin to form factor VIIIa. Factor VIIIa exists as a non-covalent dimer in a metal-linked (probably calcium) complex and functions as a cofactor in the enzymatic activation of factor X by factor IXa. Factor VIIIa is similar in structure and generation to factor Va.
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faktor X

Storage-stable glycoprotein blood coagulation factor that can be activated to factor Xa by both the intrinsic and extrinsic pathways. A deficiency of factor X, sometimes called Stuart-Prower factor deficiency, may lead to a systemic coagulation disorder.
MSH

Coagulation factor X (488 aa, 55 kDa) is encoded by the human F10 gene. This protein is involved in proteolysis and blood coagulation.
NCI

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faktor X – nedostatek

Blood coagulation disorder usually inherited as an autosomal recessive trait, though it can be acquired. It is characterized by defective activity in both the intrinsic and extrinsic pathways, impaired thromboplastin time, and impaired prothrombin consumption.
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faktor Xa

Activated form of factor X that participates in both the intrinsic and extrinsic pathways of blood coagulation. It catalyzes the conversion of prothrombin to thrombin in conjunction with other cofactors.
MSH

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faktor XI

Stable blood coagulation factor involved in the intrinsic pathway. The activated form XIa activates factor IX to IXa. Deficiency of factor XI is often called hemophilia C.
MSH

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faktor XI – nedostatek

A hereditary deficiency of blood coagulation factor XI (also known as plasma thromboplastin antecedent or PTA or antihemophilic factor C) resulting in a systemic blood-clotting defect called hemophilia C or Rosenthal`s syndrome, that may resemble classical hemophilia.
MSH

A rare inherited bleeding disorder caused by deficiency of coagulation factor XI. It may be asymptomatic or manifest with bleeding.
NCI

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faktor XIa

Activated form of factor XI. In the intrinsic pathway, Factor XI is activated to XIa by factor XIIa in the presence of cofactor HMWK; (HIGH MOLECULAR WEIGHT KININOGEN). Factor XIa then activates factor IX to factor IXa in the presence of calcium.
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faktor XII

Stable blood coagulation factor activated by contact with the subendothelial surface of an injured vessel. Along with prekallikrein, it serves as the contact factor that initiates the intrinsic pathway of blood coagulation. Kallikrein activates factor XII to XIIa. Deficiency of factor XII, also called the Hageman trait, leads to increased incidence of thromboembolic disease. Mutations in the gene for factor XII that appear to increase factor XII amidolytic activity are associated with HEREDITARY ANGIOEDEMA TYPE III.
MSH

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faktor XII – nedostatek

An absence or reduced level of blood coagulation factor XII. It normally occurs in the absence of patient or family history of hemorrhagic disorders and is marked by prolonged clotting time.
MSH

A rare autosomal recessive inherited bleeding disorder caused by deficiency of coagulation factor XII. It may be asymptomatic or manifest with bleeding.
NCI

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faktor XIIa

Activated form of factor XII. In the initial event in the intrinsic pathway of blood coagulation, kallikrein (with cofactor HIGH MOLECULAR WEIGHT KININOGEN) cleaves factor XII to XIIa. Factor XIIa is then further cleaved by kallikrein, plasmin, and trypsin to yield smaller factor XII fragments (Hageman-Factor fragments). These fragments increase the activity of prekallikrein to kallikrein but decrease the procoagulant activity of factor XII.
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faktor XIII

A fibrin-stabilizing plasma enzyme (TRANSGLUTAMINASES) that is activated by THROMBIN and CALCIUM to form FACTOR XIIIA. It is important for stabilizing the formation of the fibrin polymer (clot) which culminates the coagulation cascade.
MSH

fibrin-stabilizing plasma transglutaminase that is activated by thrombin and calcium to form factor XIIIa; important for stabilizing the formation of the fibrin polymer (clot) which culminates the coagulation cascade.
CSP

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faktor XIII – nedostatek

A deficiency of blood coagulation FACTOR XIII or fibrin stabilizing factor (FSF) that prevents blood clot formation and results in a clinical hemorrhagic diathesis.
MSH

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faktor XIIIa

Activated form of FACTOR XIII, a transglutaminase, which stabilizes the formation of the fibrin polymer (clot) culminating the blood coagulation cascade.
MSH

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fakulta

The teaching staff and members of the administrative staff having academic rank in an educational institution.
MSH

The body of teachers and administrators at a school.
NCI

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fakulta stomatologická

The teaching staff and members of the administrative staff having academic rank in a dental school.
MSH

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fakulta lékařská

The teaching staff and members of the administrative staff having academic rank in a medical school.
MSH

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fakulta ošetřovatelská

The teaching staff and members of the administrative staff having academic rank in a nursing school.
MSH

Nurse educators are registered nurses prepared at the graduate or doctoral level who prepare students for nursing practice at a variety of levels, including entry level, continuing education, advanced practice, and specialty practice. Nurse educators work in various settings, including colleges/universities, community colleges, hospitals, nursing homes, private practice, etc.
NCI

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flavinadenindinukleotid

A condensation product of riboflavin and adenosine diphosphate. The coenzyme of various aerobic dehydrogenases, e.g., D-amino acid oxidase and L-amino acid oxidase. (Lehninger, Principles of Biochemistry, 1982, p972)
MSH

coenzyme composed of riboflavin 5`-phosphate and adenosine 5`-phosphate linked by a pyrophosphate bond; it forms the prosthetic group of many flavoprotein enzymes, serving as an electron carrier by being alternately oxidized (FAD) and reduced (FMNH2); important in electron transport in mitochondria and the endoplasmic reticulum.
CSP

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Faeroereilanden

An island group between the Norwegian Sea and the North Atlantic Ocean, about one-half of the way from Iceland to Norway. (NCI)
NCI

An island group between the Norwegian Sea and the North Atlantic Ocean, about one-half of the way from Iceland to Norway. (CIA World Factbook 2002)
NCI

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HAZTEAREN ATZERAKETA/BEHAR HAINAKO DESARROILO EZA

A condition of substandard growth or diminished capacity to maintain normal function.
MSH

A clinical finding indicating less than normal growth in infancy or early childhood
NCI

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vejcovody – nemoci

Diseases involving the FALLOPIAN TUBES including neoplasms (FALLOPIAN TUBE NEOPLASMS); SALPINGITIS; tubo-ovarian abscess; and blockage.
MSH

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